THE KNEE JOINT AND ITS COMMON DISEASE.
THE
KNEE JOINT
THE
ANATOMY OF THE KNEE JOINT
THE KNEE JOINT IS
THE LARGEST JOINT IN THE HUMAN BODY.IT IS FORMED BY THREE PARTS:
1)THE LOWER END OF THE FEMUR.
2)THE UPPER END OF TIBIA.
3)THE PATELLA.
THUS, IT IS A COMPOUND JOINT. I.E, A
JOINT, WHICH IS FORMED BY UNION OF MORE THAN ONE SINGLE JOINT.
HERE TWO
COMPONENTS MAKE UP THIS JOINT.
1) TIBIO-FEMORAL(UPPER END OF TIBIA AND
LOWER END OF FEMUR) &
2) PATELLO-FEMORAL PART (A SURFACE END OF
PATELLA AND LOWER END OF FEMUR).
BASICALLY, THE KNEE JOINT IS A MODIFIED
TYPE OF HINGE JOINT.
HINGE JOINT IS A TYPE OF JOINT WHICH
PERMITS MOVEMENT IN ONE DIRECTION ONLY, i.e FLEXION AND EXTENSION, JUST LIKE A
HINGE OF A DOOR WHICH PERMITS ONLY OPENING AND CLOSING MOVEMENTS OF THE DOOR.
OTHER NAME FOR SUCH A JOINT IS GINGLYMUS
JOINT.
HOWEVER, KNEE BEING A MODIFIED COMPOUND
JOINT ALSO ALLOWS SLIGHT OUTWARD AND INWARD ROTATION ON THE AXIS APART FROM ITS
MAIN FULL FLEXION AND EXTENSION ACTION
STRUCTURES
RELATED TO KNEE JOINT
• THE KNEE JOINT CONTAINS NUMEROUS TYPES OF ATTATCHMENTS CONSISTING OF VARIOUS MUSCLES, LIGAMENTS, & CARTILAGES(MENISCUS
AND ARTICULAR CARTILAGE) . WHICH HAVE
THEIR OWN RESPECTIVE FUNCTIONS.
MUSCLES OF THE KNEE
JOINT.
1) ARTICULARIS
GENUS:
•
ORIGIN:ANTERIOR
DISTAL END OF TIBIAL SHAFT/ FRONT LOWER END OF TIBIA.
•
INSERTION:ON THE
SYNOVIAL MEMBRANE OF THE KNEE JOINT.
•
ACTION: IT PULLS THE
SUPRA-PATELLAR BURSA,.i.e. A FLUID FILLED BAG LIKE STRUCTURE (BURSA), LOCATED
ABOVE THE PATELLA(KNEE CAP), DURING EXTENSION OF KNEE JOINT, TO PREVENT INJURY
TO THE KNEE BY REDUCING FRICTION.
ARTICULARIS GENU MUSCLE
1)THE QUADRICPES
•
THIS MUSCLE HAS 4 MUSCLE HEADS AS THE NAME SUGGESTS(QUAD)
1. RECTUS FEMORIS, COVERING THE FRONT OF THE THIGH.
2. VASTUS LATERALIS: LOCATED ON OUTER THIGH.
3. VASTUS MEDIALIS: INNER THIGH.
4. VASTUS
INTERMEDIUS: INFRONT OF THE THE THIGH BEHIND RECTUS FEMORIS.
FUNCTION:STRONG EXTENSION OF THE KNEE.
LIGAMENTS OF THE
KNEES
THE
LIGAMENTS OF THE KNEE JOINT ARE CLASSIFIED BY THEIR LOCATION, USING THE
SYNOVIAL CAPSULE OF THE KNEE AS A REFERENCE. THUS, THE TYPES OF LIGAMENTS OF
THE KNEES CAN BE DIVIDED INTO TWO PARTS.
1.
INTRACAPSULAR LIGMENTS.(INSIDE THE SYNOVIAL CAPSULE)
2.
EXTRACAPSULAR LIGAMENTS.(OUTSIDE THE SYNOVIAL
CAPSULE)
1). EXTRACAPSULAR LIGAMENTS:
•
MEDIAL COLLATERAL LIGAMENT(M.C.L).
•
FUNCTION:PREVENT EXCESSIVE
OUTWARD/EXTERNAL ROTATION OF THE JOINT
•
LATERAL COLLATERAL LIGAMENT(L.C.L)
•
FUNCTION:PREVENT
EXCESSIVE INWARDS/INTERNAL ROTATION OF THE JOINT
2). INTRACAPSULAR LIGAMENT:
•
ANTERIOR CRUCIATE LIGAMENT( A.C.L):
FUNCTION: PREVENTS OVERFLEXION OF THE KNEE.
•
POSTERIOR CRUCIATE LIGAMENT(L.C.):
FUNCTION: PREVENTS OVEREXTENSION OF THE KNEE.
BOTH THESE
LIGAMENTS TOGETHER FORM AN ‘X’ (CROSS) LIKE ARRANGEMENT.HENCE, THEY ARE TERMED
AS CRUCIATE.
LIGAMENTS
OF THE KNEE JOINT
THE
SYNOVIAL CAPSULE
•
THIS IS A THIN LAYER OF FIBROUS TISSUE SURROUNDING
THE KNEE JOINT. THE CAVITY FORMED IS KNOWN AS SYNOVIAL MEMBRANE WHICH BEARS
SYNOVIAL FLUID.
•
THE MAIN FUNCTION OF THE SYNOVIAL CAPSULE IS TO
PROTECT THE KNEE JOINT. THE SYNOVIAL MEMBRANE INSIDE THE CAPSULE SECRETES THE SYNOVIAL FLUID IN THE SYNOVIAL
CAVITY. THE SYNOVIAL FLUID INSIDE THE SYNOVIAL MEMBRANE, IS A FILTRATE OF
PLASMA AND ALSO THE PROTEINS PRODUCED BY JOINT TISSUES. BASICALLY THE FLUID
CONTAINS HYALURONAN(FIBROBLAST LIKE CELLS IN SYNOVIUM) AND LUBRICAN
PROTEOGLYCANS(CHONDROCYTES OR CARTILAGE CELLS).
•
THIS SYNOVIAL FLUID ALSO CREATES A MEMBRANE WHICH IS
ADSORBED ON THE CARTILAGE, WHICH WHEN EXPOSED TO MOVEMENT SQUEEZES OUT SYNOVIAL
FLUID TO REDUCE THE DAMAGE TO THE CARTILAGE AND ALSO FACILITATE SMOOTH
MOVEMENT.
•
THE SYNOVIAL FLUID HELPS IN REDUCTION OF FRICTION,
SHOCK ABSORPTION AND PROVIDING NUTRITION TO THE JOINT.
COMMONEST PROBLEMS AFFECTING THE KNEES
•
KNEES BEING THE MOST WEIGHT BEARING PARTS, ARE
CONTINUOUSLY SUBJECTED TO WEAR AND TEAR, HERE ARE THE COMMONEST PROBLEMS WHICH
INVOLVE THE KNEE.
LIGAMENT INJURY/
SPORTS INJURY
•
SPRAINS
•
M.C.L AND L.CL. SPRAIN ARE THE COMMONEST INJURY, SPECIALLY IN ATHLETES. IT CAN OCCUR DUE TO A
SUDDEN OUTWARD THRUST(L.C.L INJURY), OR SUDDEN INWARD THRUST(M.C.L INJURY) OR
MISSTEP
•
•
SYMPTOMS:
•
DEPENDING UPON THE LIGAMENT INVLOVED.
•
L.C.L SPRAIN:PAIN IN
THE OUTER/LATERAL PART OF THE KNEE, WITH OR WITHOUT TENDERNESS. PAIN SPECIALLY
FELT OF TURNING THE FOOT OUTWARD.
M.C.L SPRAIN: PAIN IN THE INNER/MEDIAL PART OF KNEE, WITH OR
WITHOUT TENDERNESS. PAIN SPECIALLY FELT OF TURNING THE FOOT INWARDS
· A.C.L & P.C.L
INJURY: ANTERIOR CRUCIATE LIGAMENT AND POSTERIOR CRUCIATE LIGAMENT INJURY AND
SPRAIN ARE ALSO VERY COMMON.
COMMONLY FOUND IN ATHLETES, THIS SPRAIN IS CAUSED BY SIDDEN OVEREXTENSION(P.C.L
SPRAIN) OR SUDDEN OVER FLEXION (A.C.L SPRAIN).
SYMPTOMS INCLUDE:
EXCRUCIATING PAIN INSIDE THE HOLLOW OF
THE KNEE TO AN EXTENT THAT PREVENTS MOVEMENT. SWELLING AND TENDERNESS OF THE KNEE. WITH SENSE OF IMPROPER
ARTICULATION/GIVE IN SENSATION, MADE WORSE BY WALKING.
MANY A TIMES, IF THE BLOW IS SEVERE
ENOUGH, IT CAN LEAD TO TOTAL DAMAGE TO THE LIGAMENTS WITHIN, LEADING TO
LIGAMENTAL TEAR. MOST COMMON LIGAMENTAL TEARS INVOLVE ANTERIOR CRUCIATE
LIGAMENT TEAR AND POSTERIOR CRUCIATE LIGAMENT TEAR.
IF THE BLOW IS SEVERE ENOUGH IT CAN
CAUSE A.C.L OR P.C.L OR SOMETIMES COMBINED BUCKLING OF BOTH TOGETHER.
OSTEOARTHRITIS
OF THE KNEE
• BY FAR THE MOST COMMONEST DISEASE INVOLVING THE KNEE JOINT,
• OSTEOARTHRITIS IS A DEGENERATIVE DISEASE OF THE JOINT. IN THIS CONDITION
THERE IS A GRADUAL DEGRADATION OF THE CARTILAGE AND REDUCTION IN JOINT
SPACE.ULTIMATELY THE ARTICULAR SURFACES GET EXPOSED AND FRICTION BETWEEN THE
TWO SURFACES LEAD TO PRODUCTION OF CALCIUM SPIKES KNOWN AS ‘OSTEOPHYTIC SPURS’.
• CAUSES
AND RISK FACTORS OF OA
• PRIMARY: WHERE NO SPECIFIC CAUSE IS FOUND, AND FAMILIAL TENDENCY AND AGE
FACTORS INCREASE THE CHANCES OF DEVELOPING O.A., HOWEVER OBESITY AND FAMILIAL
BACKGROUND INCREASE THE SUSCEPTIBILITY OF DEVELOPING IT.
• OVEREXERTION LIKE LIFTING HEAVIER WEIGHTS, CONTINUOUS EXERTION LIKE STAIR
CLIMBING AND WORKPLACE ETC.
• SECONDARY CAUSES:
• DIABETES MELLITUS.
• HAEMOCHROMATOSIS AND WILSON DISEASE.
• MARFANS SYNDROME.
• JOINT INFECTION.
• EHLER-DANLOS SYNDROME.
• PERTH’S DISEASE.
• AUTOIMMUNE DISEASE:RA AND GOUT.
•
SIGNS AND SYMPTOMS:
•
PAIN AND STIFFNESS ARE THE CHIEF COMPLAINTS IN
PATIENTS SUFFERING FROM OA.
•
PAINS ARE MADE WORSE ON PRLONGED EXERTION AND
IMPROVE BY REST.BUT LATER ON, AS THE DISEASE PROCEEDS WITH WORSENING PATHOLOGY
AND JOINT SPACE GOES ON REDUCING, THE PERSON MAY EXPERIENCE SEVERE PAIN ON THE
SLIGHTEST MOVEMENTS.
•
THE JOINT MAY PRODUCE A TYPICAL CRACKLING
SOUND(CREPITUS) WHEN MOVED.
CLASSIFICATION
OF OA:
•
OSTEOARTHRITIS, IN GENERAL, CAN BE CLASSIFIED IN
VARIOUS WAYS:
•
DEPENDING ON THE CAUSE:1)PRIMARY O.A.2) SECONDARY O.A.
•
WOMAC(Western Ontario and McMaster Universities
Osteoarthritis Index):PAIN, STIFFNESS
AND FUNCTIONAL LIMITATIONS.
•
Kellgren-Lawrence grading scale FOR OSTEOARTHRITIS OF KNEE DIAGNOSIS IS DONE BY 2D RADIO IMAGING.
THE ROLE OF HOMOEOPATHY IN KNEE JOINT
TROUBLES.
HOMOEOPATHY IN KNEE TROUBLES, SPECIALLY
OSTEO ARTHRITIS, NOT ONLY REDUCES PAIN, SWELLING AND RESTRICTED MOVEMENTS, BUT
ALSO IMPROVES THE PATHOLOGY WITHIN.THEREBY COMPLETELY RESTORING THE PATIENT TO
NORMAL.
THOUGH A CONSTITUTIONAL APPROACH IS
THE BEST IN RESTORING THE PATIENT SOME IMPORTANT REMEDIES ARE AS FOLLOWS.
1.
ARNICA
2. BELLIS PER
3. RHUS TOX
4. CALCAREA CARB
5. CALCAREA FLOUR
6. CALCAREA PHOS
7. RUTA G
8. STONTIUM CARB
9. NATRUM MUR
10. LEDUM PAL AND
MANY MORE….
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